| viXra.org > Biochemistry > 2402 |
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| viXra.org > Biochemistry > 2402 |
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[2] viXra:2402.0062 [pdf] submitted on 2024-02-12 04:06:57
Authors: Reginald B. Little
Comments: 86 Pages. Author develops more details of his theory of induced nuclear magnetic moments for life.
This review highlights the author’s theory of quantum fluctuations in systems down to nuclei, nucleons and quarks (as induced by surrounding thermal space, gravity, electric and magnetic fields and other nucleons) releasing and/or inducing nuclear fields, nuclear magnetic moment and/or spins. Many isotopes of elements are presented to give proof by many correlations of novel physical, chemical, biological, nuclear, thermodynamic and transport properties with nonzero nuclear magnetic moments (NMMs) and spins. The author further develops his theory of such fractional reversible (FR) fissing and fusing by perpetual quantum fluctuations of quarks and nucleons correlating with proximity of nucleons to magic number (MN) nuclei (2, 8, 20, 28, 50, 82, 126 u2026) and magic number nucleonic molecules (MNNMs). By such the author is able to correlate stable isotopes of various elements and their zero, nonzero and chiral NMMs to their masses relative MNs. The lifetimes of unstable isotopes are also reasoned and correlated to relative separation from MNs. The stable and unstable nuclei for various isotopes are presented by the authors theory to undergo hidden FR transmutations by FR electron capture (EC), FR electron release (ER), FR proton and neutron captures and/or releases as reasoned by infinitesimal quantum fluctuations of nuclei by the author’s theory. New catalytic system of Ne solvent with NaF solute is introduced in highly activated conditions for catalyzing CO2 + H2O ↔ CxHy and CxHyOz by NMMs and induced NMMs in the catalytic solute and solvent Ne and NaF. By such correlated phenomena, the author develops more his theory of new physicochemical basis for life; novel catalysis and enzymatics, unconventional nuclear reactions, room temperature and pressure superconductivity, and gravitational chemistry.
Category: Biochemistry
[1] viXra:2402.0018 [pdf] replaced on 2024-02-11 15:45:11
Authors: Patrick Douglas Shaw Stewart
Comments: 16 Pages.
Several recent studies conclude that an increase in the pathogenicity of SARS-CoV-2 cannot be ruled out. However, this does not mean that increased pathogenicity is probable. Note that SARS-CoV-2 is a "direct" respiratory virus - meaning it is usually spread by the respiratory route but does not routinely pass through the lymphatics like measles and smallpox. Moreover, it is not normally spread by the fecal-oral route. Providing its tropism does not change, it will be unique if its pathogenicity does not decrease over time until it becomes similar to influenza and common cold viruses. As pointed out by Ewald in the 1980s, most respiratory viruses, unlike many other viruses, benefit greatly from the mobility of their hosts, creating a strong selective pressure favoring respiratory virus strains that cause only mild symptoms. In this review, I discuss the biological factors that tend to reduce the pathogenicity of respiratory viruses, including sensitivity to heat (which means the virus is less likely to multiply in warmer areas such as the lungs) and alterations to the cleavage sites and binding domains on the virus's surface proteins. Increasing immunity within a host population may increase selection for mild strains. Conversely, several biological factors could potentially increase respiratory viruses’ pathogenicity. These include the virus replicating in the lymphatic system, spreading via solid surfaces, and transmitting through fecal matter, including contaminated water. Furthermore, human activities and political events could increase the harmfulness of SARS-CoV-2. These include the following: large-scale testing, especially by methods where the test results are delayed by one or more days, such as PCR tests; the spread of the virus in settings where people are close together and not free to move around; poor hygiene facilities in developing countries; and social, political, or cultural influences that encourage people to remain socially active when they are sick, including political crises such as wars. If we can avoid these and similar eventualities, it is likely that SARS-CoV-2 will evolve to become milder, eventually becoming another common cold virus. However, the timescale is uncertain. Observations of previous influenza and influenza-like pandemics suggest that in the absence of effective medical interventions, it may take two or three decades for the virus to become indistinguishable from other cold viruses.
Category: Biochemistry
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